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- W2393960958 abstract "Objective:To observe the role of sFKN and TNF-α in coronary heart disease and their changes after Benazepril treatment.Methods:There were 88 people enrolled in this study, including 15 normal controls, 25 stable angina (SA) patients and 48 acute coronary syndrom (ACS) patients. ACS patients were then divided into two groups according to Benazepril treatment.The levels of serum soluble Fractalkine (sFKN) and TNF-α were measured by enzyme linked immunosorbent assay (ELISA), and the correlation between these 2 indexes and Gensini score was investigated in all of those groups. Results:Compared to the control group, the level of serum sFKN was significantly increased in ACS group (P0.01), and there was no significant difference between control group and SA group. Both of the levels of serum TNF-αwere markedly increased in ACS and SA groups, especially the former gourp, compared to those in controls (P0.01). Additionally, the levels of these 2 indexes in ACS patients treated with Benazepril were higher than those with no Benazepril treatment (P0.05). Liner regression showed sFKN was positively correlated to TNF-α (r=0.7), and there was no correlation found between these 2 indexes and Gensini score.Conclusion:TNF-α and sFKN played an important role in the development of coronary heart disease, and Benazepril could improve the disease by inhibiting these 2 cytokines." @default.
- W2393960958 created "2016-06-24" @default.
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- W2393960958 date "2010-01-01" @default.
- W2393960958 modified "2023-09-23" @default.
- W2393960958 title "The role of sFKN and TNF-α in coronary heart disease and their changes after treatment with Benazepril" @default.
- W2393960958 hasPublicationYear "2010" @default.
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