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• W2395922267 abstract "Thrombotic microangiopathies (TMAs) include thrombotic thromobocytopenic purpura and hemolytic uremic syndrome (HUS). Among these conditions, atypical HUS is now recognized to be a disease of alternative complement pathway dysregulation. Eculizumab is a recombinant humanized monoclonal antibody that binds to the complement protein C5 and prevents the cleavage of C5 to C5a and C5b. Eculizumab has been used as a novel treatment for complement-mediated TMA. We present a case of a patient with human immunodeficiency virus infection who developed TMA and was successfully treated with eculizumab. The effect of long-term treatment with this new medication is unknown, and further studies are needed to establish guidelines in the management of this condition." @default.
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• W2395922267 date "2016-05-17" @default.
• W2395922267 modified "2023-09-29" @default.
• W2395922267 title "Thrombotic microangiopathy and human immunodeficiency virus in the era of eculizumab" @default.
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• W2395922267 doi "https://doi.org/10.1093/ckj/sfw035" @default.
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