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- W2396670243 endingPage "2086" @default.
- W2396670243 startingPage "2075" @default.
- W2396670243 abstract "Toll-like receptors (TLRs), well-characterized pattern-recognizing receptors of the innate arm of the immune system, are vital in detecting pathogen-associated molecular patterns (PAMPs). The TLR-PAMP interaction initiates an intracellular signaling cascade, predominantly culminating in upregulation of antiviral components, including inducible nitric oxide synthase (iNOS). After activation, various TLR pathways can promote iNOS production via the myeloid differentiation primary response-88 (MyD-88) adapter protein. Subsequently, iNOS facilitates production of nitric oxide (NO), a highly reactive and potent antiviral molecule that can inhibit replication of RNA and DNA viruses. Furthermore, NO can diffuse freely across cell membranes and elicit antiviral mechanisms in various ways, including direct and indirect damage to viral genomes. This review emphasizes current knowledge of NO-mediated antiviral responses elicited after activation of TLR signaling pathways." @default.
- W2396670243 created "2016-06-24" @default.
- W2396670243 creator A5010407946 @default.
- W2396670243 creator A5040164160 @default.
- W2396670243 creator A5043250679 @default.
- W2396670243 date "2016-05-27" @default.
- W2396670243 modified "2023-10-14" @default.
- W2396670243 title "Activation of toll-like receptor signaling pathways leading to nitric oxide-mediated antiviral responses" @default.
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