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• W2397133092 endingPage "1706" @default.
• W2397133092 startingPage "1701" @default.
• W2397133092 abstract "We studied the effects of adding washed human platelets or platelets with nonintact glutathione redox cycles to endothelial cell monolayers treated with glucose oxidase to initiate oxidant stress and increase permeability. Changes in 125I-labeled albumin transmonolayer movement were used as the index of monolayer permeability. Washed human platelets attenuated oxidant-induced increases in albumin flux. Platelets treated with 1,3-bis(2-chloroethyl)-1-nitrosurea, 1-chloro-2,4-dinitrobenzene, or buthionine sulfoximine to inhibit selective enzymatic steps in the glutathione redox cycle decreased permeability to a lesser degree. We conclude that 1) washed human platelets attenuate monolayer permeability defects in aortic endothelial monolayers exposed to glucose oxidase and 2) the protective effects of platelets are partially dependent on an intact platelet glutathione redox cycle." @default.
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• W2397133092 date "1996-10-01" @default.
• W2397133092 modified "2023-10-18" @default.
• W2397133092 title "Platelets attenuate oxidant-induced permeability in endothelial monolayers: glutathione-dependent mechanisms" @default.
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• W2397133092 doi "https://doi.org/10.1152/jappl.1996.81.4.1701" @default.
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