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- W2397367056 abstract "Ingested food antigens rapidly cross the gastrointestinal barrier and reach pro-inflammatory cells in the skin. Food allergy provokes urticaria/angioedema by classical, Type I, IgE-mediated hypersensitivity. Food-induced atopic dermatitis is the result of non-classical, IgE-directed hypersensitivity involving resident mast cells, Langerhans cells, CD4+, TH2 lymphocytes and monocytes. A form of gluten sensitivity provokes a characteristic eczematous-like rash and enteropathy (Dermatitis herpetiformis)." @default.
- W2397367056 created "2016-06-24" @default.
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- W2397367056 date "1995-01-01" @default.
- W2397367056 modified "2023-09-23" @default.
- W2397367056 title "Mechanisms in adverse reactions to food. The skin." @default.
- W2397367056 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/7741202" @default.
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