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- W2400985808 abstract "F 51 Hypercholesterolaemia, Flail septum 16 mm Amplatzer ASO hypertension M 42 – Flail septum 20 mm Helex F 48 Hypercholesterolaemia, PFO only 10 mm Amplatzer ASO obese, smoker M 39 – Flail septum 12 mm Amplatzer ASO P aradoxical embolism due to the presence of patent foramen ovale (PFO) is an accepted mechanism of ischaemic cryptogenic stroke. Transcatheter closure of the patent foramen is the logical, safe and accepted technique for the prevention of further neurological episodes in individuals who have had transient ischaemic events or stroke. Complications are infrequent and arrhythmias are occasionally mentioned in large series. We report four individuals (out of a total of 96 atrial septal defect/PFO closures) who developed transient atrial fibrillation (AF) 4-14 days after device closure. All four had initially presented with neurological events, were documented as having PFOs on echocardiography with shunt reversal, had uneventful PFO device closures, and continued on 6 months of daily, low dose aspirin. Patient characteristics are given in Table 1. Presentation in AF was with varying combinations of palpitations, fatigue, shortness of breath, dizziness and headaches. AF was easily controlled with cardioversion (two required cardioversion twice), and patients were loaded with amiodarone, which was continued orally for 3 months. Atrial Fibrillation Following Transcatheter Closure of Patent Foramen Ovale" @default.
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- W2400985808 date "2008-03-21" @default.
- W2400985808 modified "2023-09-23" @default.
- W2400985808 title "Atrial fibrillation following transcatheter closure of patent foramen ovale." @default.
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