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- W2406906900 abstract "Experiments on the frog sartorius muscle showed that nonhydrolisable acetylcholine analog carbachol (CCh) depresses spontaneous quantal mediator release via muscarinic M2 receptors of nerve ending. Adenosine (Ade) acting via inhibitory A1 receptors is another strong spontaneous quantal release modulator. Inhibition of pertussis toxin (PTx)-sensitive G-proteins only partly eliminated CCh and Ade depressive action. It means metabotropic A1 and M2 receptors of the frog nerve ending regulate spontaneous quantal release via activating of both PTx-sensitive and PTx-insensitive inhibitory mechanisms." @default.
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- W2406906900 date "2006-06-01" @default.
- W2406906900 modified "2023-09-23" @default.
- W2406906900 title "Mechanism of carbachol and adenosine action on spontaneous quantal mediator release at frog neuromuscular junction" @default.
- W2406906900 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16967873" @default.
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