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- W2407170691 abstract "The present study was designed to assess the roles of activation and arachidonic acid (AA) metabolites in hypoxia-induced renal cell injury. Hypoxia increased LDH release in a dose-dependent manner in rabbit renal cortical slices, and this increase was significant after 20-min hypoxia. The hypoxia-induced LDH release was prevented by amino acids, glycine and alanine, and extracellular acidosis (pH 6.0). Buffering intracellular by a chelator, but not omission of in the medium produced a significant reduction in hypoxia-induced LDH release. The effect of hypoxia was blocked by inhibitors, mepacrine, butacaine, and dibucaine. A similar effect was observed by a 85-kD inhibitor AA increased hypoxia-induced LDH release, and albumin, a fatty acid absorbent, prevented the LDH release, suggesting that free fatty acids are involved in hypoxia-induced cell injury. These results suggest that activation and its metabolic products play important roles in pathogenesis of hypoxia-induced cell injury in rabbit renal cortical slices." @default.
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- W2407170691 date "1999-01-01" @default.
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- W2407170691 title "Role of Phospholipase $A_2$ in Hypoxia-Induced Renal Cell Injury" @default.
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