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• W2413667918 abstract "In this chapter, we describe model systems to study leukemia driven by the Abelson oncogene. In people, the Abelson oncogene results from the chromosomal translocation t(9;22)(q34;q11) that is found in more than 90 % of all human chronic myeloid leukemia (CML) patients and in 20-25 % of patients suffering from acute lymphoid leukemia (ALL). This translocation is also called Philadelphia chromosome and encodes the BCR/ABL oncogene, a constitutive active tyrosine kinase. BCR/ABL renders hematopoietic cells independent from exogenous growth-stimulatory signals by continuously engaging signaling pathways including JAK-STAT signaling and the MAPK pathway. The enforced expression of BCR/ABL suffices to transform hematopoietic cells which made it to one of the best studied model systems in the field. Here we present methods to study BCR/ABL-triggered leukemia and solid lymphoid tumor formation." @default.
• W2413667918 created "2016-06-24" @default.
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• W2413667918 date "2015-01-01" @default.
• W2413667918 modified "2023-09-27" @default.
• W2413667918 title "Modeling BCR/ABL-Driven Malignancies in the Mouse" @default.
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• W2413667918 doi "https://doi.org/10.1007/978-1-4939-2297-0_12" @default.
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