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- W2414515032 endingPage "887" @default.
- W2414515032 startingPage "887" @default.
- W2414515032 abstract "Heart failure (HF) is a major public health priority due to its epidemiological transition and the world's aging population. HF is typified by continuous loss of contractile function with reduced, normal, or preserved ejection fraction, elevated vascular resistance, fluid and autonomic imbalance, and ventricular dilatation. Despite considerable advances in the treatment of HF over the past few decades, mortality remains substantial. Pharmacological treatments including β-blockers, angiotensin-converting enzyme inhibitors, angiotensin receptor blockers, and aldosterone antagonists have been proven to prolong the survival of patients with HF. However, there are still instances where patients remain symptomatic, despite optimal use of existing therapeutic agents. This understanding that patients with chronic HF progress into advanced stages despite receiving optimal treatment has increased the quest for alternatives, exploring the roles of additional pathways that contribute to the development and progression of HF. Several pharmacological targets associated with pathogenesis of HF have been identified and novel therapies have emerged. In this work, we review recent evidence from proposed mechanisms to the outcomes of experimental and clinical studies of the novel pharmacological agents that have emerged for the treatment of HF." @default.
- W2414515032 created "2016-06-24" @default.
- W2414515032 creator A5027013608 @default.
- W2414515032 creator A5029328435 @default.
- W2414515032 creator A5034257019 @default.
- W2414515032 creator A5043622638 @default.
- W2414515032 creator A5052177400 @default.
- W2414515032 date "2016-06-01" @default.
- W2414515032 modified "2023-10-02" @default.
- W2414515032 title "Review of novel therapeutic targets for improving heart failure treatment based on experimental and clinical studies" @default.
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