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- W2419095158 endingPage "e1171284" @default.
- W2419095158 startingPage "e1171284" @default.
- W2419095158 abstract "Autophagy is a cell survival mechanism by which bulk cytoplasmic material, including soluble macromolecules and organelles, is targeted for lysosomal degradation. The role of autophagy in diverse cellular processes such as metabolic stress, neurodegeneration, cancer, aging, immunity, and inflammatory diseases is being increasingly recognized. Epithelial cell junctions play an integral role in the cell homeostasis via physical binding, regulating paracellular pathways, integrating extracellular cues into intracellular signaling, and cell-cell communication. Recent data indicates that cell junction composition is very dynamic. The junctional protein complexes are actively regulated in response to various intra- and extra-cellular clues by intracellular trafficking and degradation pathways. This review discusses the recent and emerging information on how autophagy regulates various epithelial cell junctions. The knowledge of autophagy regulation of epithelial junctions will provide further rationale for targeting autophagy in a wide variety of human disease conditions." @default.
- W2419095158 created "2016-06-24" @default.
- W2419095158 creator A5041513548 @default.
- W2419095158 creator A5090149192 @default.
- W2419095158 date "2016-06-09" @default.
- W2419095158 modified "2023-10-14" @default.
- W2419095158 title "Role of autophagy in the regulation of epithelial cell junctions" @default.
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