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• W2440773062 abstract "This study examined the effect of high-intensity interval training on the V ˙ O 2 response during severe, constant-load exercise. Prior to, and following training, 10 females ( V ˙ O 2 peak 37.4 ± 6.0 mL kg−1 min−1) performed a graded exercise test to determine V ˙ O 2 peak and lactate threshold (LT) and a 6 min cycle test (CT) at the pre-training V ˙ O 2 peak intensity. Training involved high-intensity intervals (2 min work, 1 min rest) performed 3× week for 8 weeks. Breath-by-breath data from 0 to 6 min during the CT were smoothed using 5 s averages and fit to a bi-exponential model starting from 20 s. Training resulted in significant improvements in V ˙ O 2 max (2.34 ± 0.37–2.78 ± 0.30 L min−1), power at V ˙ O 2 max (170 ± 26–204 ± 25 W) and power at LT (113 ± 17–136 ± 20 W) (p < 0.05). Following training, the V ˙ O 2 response showed a significant increase in the amplitude of the primary phase (A1) (1396 ± 103–1695 ± 100 mL min−1; p < 0.05) and end-exercise V ˙ O 2 ( V ˙ O 2 EE ), with no difference (p > 0.05) in the time constants of either phase or the amplitude of the slow component (318 ± 67–380 ± 48 mL; p = 0.15). In conjunction, accumulated oxygen deficit (AOD) (43.7 ± 9.8–17.2 ± 2.8 mL O2 eq kg−1) and anaerobic contribution to the CT (19.4 ± 4.4–7.2 ± 1.2%) were significantly reduced. In contrast to previous moderate-intensity research, a high-intensity interval training program increased A1 and V ˙ O 2 EE for the same absolute exercise intensity, decreasing the AOD during a severe-intensity CT. This study examined the effect of high-intensity interval training on the V ˙ O 2 response during severe, constant-load exercise. Prior to, and following training, 10 females ( V ˙ O 2 peak 37.4 ± 6.0 mL kg−1 min−1) performed a graded exercise test to determine V ˙ O 2 peak and lactate threshold (LT) and a 6 min cycle test (CT) at the pre-training V ˙ O 2 peak intensity. Training involved high-intensity intervals (2 min work, 1 min rest) performed 3× week for 8 weeks. Breath-by-breath data from 0 to 6 min during the CT were smoothed using 5 s averages and fit to a bi-exponential model starting from 20 s. Training resulted in significant improvements in V ˙ O 2 max (2.34 ± 0.37–2.78 ± 0.30 L min−1), power at V ˙ O 2 max (170 ± 26–204 ± 25 W) and power at LT (113 ± 17–136 ± 20 W) (p < 0.05). Following training, the V ˙ O 2 response showed a significant increase in the amplitude of the primary phase (A1) (1396 ± 103–1695 ± 100 mL min−1; p < 0.05) and end-exercise V ˙ O 2 ( V ˙ O 2 EE ), with no difference (p > 0.05) in the time constants of either phase or the amplitude of the slow component (318 ± 67–380 ± 48 mL; p = 0.15). In conjunction, accumulated oxygen deficit (AOD) (43.7 ± 9.8–17.2 ± 2.8 mL O2 eq kg−1) and anaerobic contribution to the CT (19.4 ± 4.4–7.2 ± 1.2%) were significantly reduced. In contrast to previous moderate-intensity research, a high-intensity interval training program increased A1 and V ˙ O 2 EE for the same absolute exercise intensity, decreasing the AOD during a severe-intensity CT." @default.
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• W2440773062 date "2006-06-01" @default.
• W2440773062 modified "2023-09-23" @default.
• W2440773062 title "Effects of high-intensity interval training on the response during severe exercise" @default.
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• W2440773062 doi "https://doi.org/10.1016/j.jsams.2006.03.014" @default.
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