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• W2441428989 abstract "Exposure to ultraviolet (UV) light is closely related to human diseases, such as skin cancer, due to irreversible injuries to the skin cells. The UV-induced DNA damage and programmed cell death are important determinants for skin carcinogenesis. The aim of the present study was to investigate the anti-ultraviolet-C (UVC) effects of pyridoxamine in human keratinocyte HaCaT cells and its mechanisms of action.UVC-induced programmed cell death in HaCaT cells was abrogated by treated the cells immediately after UVC irradiation with 40, 80 and 160 μM of pyridoxamine. Monitoring the UVC-induced-specific reactive oxygen species, we found that 20, 40, 80 and 160 μM of pyridoxamine was also effective in suppressing the induction of reactive oxygen species by UVC.Overall, our results provided evidence showing that pyridoxamine was effective in protecting HaCaT cells from UVC-induced programmed cell death and may be a potential anti-UVC agent in life and clinical practice." @default.
• W2441428989 created "2016-06-24" @default.
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• W2441428989 date "2015-05-16" @default.
• W2441428989 modified "2023-09-27" @default.
• W2441428989 title "Protective Effects of Pyridoxamine Against UVC-induced Programmed Cell Death in HaCaT Cells." @default.
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• W2441428989 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/25977384" @default.
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