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- W2463996251 abstract "Spasticity refers to an abnormal increase in muscle tone characterized by velocity-dependent hyperexcitability of the spinal stretch reflexes. Both intraspinal processing and supraspinal control are important in the regulation of muscle tone [ 1 Li S. Francisco G.E. New insights into the pathophysiology of post-stroke spasticity. Front. Hum. Neurosci. 2015; 9: 192 Crossref PubMed Scopus (120) Google Scholar , 2 Mukherjee A. Chakravarty A. Spasticity mechanisms — for the clinician. Front. Neurol. 2010; 1: 149 Crossref PubMed Scopus (143) Google Scholar ]. The role of descending tracts from the brainstem, such as the dorsal reticulospinal tract, is emphasized [ 1 Li S. Francisco G.E. New insights into the pathophysiology of post-stroke spasticity. Front. Hum. Neurosci. 2015; 9: 192 Crossref PubMed Scopus (120) Google Scholar , 2 Mukherjee A. Chakravarty A. Spasticity mechanisms — for the clinician. Front. Neurol. 2010; 1: 149 Crossref PubMed Scopus (143) Google Scholar ]. Although impaired dexterity and hypotonia are considered the main sequelae of isolated lesions in the pyramidal tract (PRT), some studies have also described the development of spasticity in association with isolated PRT lesions [ 3 Growdon J.H. Chambers W.W. Liu C.N. An experimental study of cerebellar dyskinesia in the rhesus monkey. Brain. 1967; 90: 603-632 Crossref PubMed Scopus (43) Google Scholar , 4 Paulson G.W. Yates A.J. Paltan-Ortiz J.D. Does infarction of the medullary pyramid lead to spasticity?. Arch. Neurol. 1986; 43: 93-95 Crossref PubMed Scopus (28) Google Scholar ]." @default.
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- W2463996251 date "2016-09-01" @default.
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- W2463996251 title "Spasticity secondary to isolated involvement of the pyramidal tract" @default.
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- W2463996251 doi "https://doi.org/10.1016/j.jns.2016.06.072" @default.
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