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- W2467079582 abstract "Glucocorticoids (GCs) regulate distinct physiological processes in the developing fetus, in particular accelerating organ maturation that enables the fetus to survive outside the womb. In preterm birth, the developing fetus does not receive sufficient exposure to endogenous GCs in utero for proper organ development predisposing the neonate to complications including intraventricular hemorrhage, respiratory distress syndrome (RDS) and necrotizing enterocolitis (NEC). Synthetic GCs (sGCs) have proven useful in the prevention of these complications since they are able to promote the rapid maturation of underdeveloped organs present in the fetus. While these drugs have proven to be clinically effective in the prevention of IVH, RDS and NEC, they may also trigger adverse developmental side effects. This review will examine the current clinical use of antenatal sGC therapy in preterm birth, their placental metabolism, and their effects on the developing brain." @default.
- W2467079582 created "2016-07-22" @default.
- W2467079582 creator A5037723145 @default.
- W2467079582 creator A5066345618 @default.
- W2467079582 creator A5070975340 @default.
- W2467079582 creator A5084366248 @default.
- W2467079582 date "2016-10-01" @default.
- W2467079582 modified "2023-10-14" @default.
- W2467079582 title "Effects of antenatal glucocorticoids on the developing brain" @default.
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- W2467079582 doi "https://doi.org/10.1016/j.steroids.2016.05.012" @default.
- W2467079582 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5052110" @default.