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- W2471125040 endingPage "141" @default.
- W2471125040 startingPage "131" @default.
- W2471125040 abstract "Many aspects of human health and disease display daily rhythmicity. The brain’s suprachiasmic nucleus, which interprets recurring external stimuli, and autonomous molecular networks in peripheral cells together, set our biological circadian clock. Disrupted or misaligned circadian rhythms promote multiple pathologies including chronic inflammatory and metabolic diseases such as atherosclerosis. Here, we discuss studies suggesting that circadian fluctuations in the vessel wall and in the circulation contribute to atherogenesis. Data from humans and mice indicate that an impaired molecular clock, disturbed sleep, and shifting light–dark patterns influence leukocyte and lipid supply in the circulation and alter cellular behavior in atherosclerotic lesions. We propose that a better understanding of both local and systemic circadian rhythms in atherosclerosis will enhance clinical management, treatment, and public health policy." @default.
- W2471125040 created "2016-07-22" @default.
- W2471125040 creator A5034311334 @default.
- W2471125040 creator A5063211617 @default.
- W2471125040 date "2016-06-24" @default.
- W2471125040 modified "2023-10-13" @default.
- W2471125040 title "Circadian Influence on Metabolism and Inflammation in Atherosclerosis" @default.
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