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• W2472522918 abstract "Mammals harbor a complex gut-associated microbiota, comprising bacteria that provide immunological, metabolic and neurological benefits to the host, and contribute to their well-being. However, dysregulation of the microbiota composition, known as dysbiosis, along with the associated mucosal immune response have a key role in the pathogenesis of many inflammatory diseases, including inflammatory bowel diseases (IBDs), type 1 and type 2 diabetes, asthma, multiple sclerosis, among others. In addition, outside the gut lumen, bacteria from microbiota are the causative agent of peritoneal inflammation, abdominal sepsis and systemic sepsis. Critical care interventions during sepsis by antibiotics induce dysbiosis and present acute and long-term poor prognosis. In this review, we discuss immunomodulatory effects of the microbial molecules and products, highlighting the role of Bacteroides fragilis, a human commensal with ambiguous interactions with the host. Moreover, we also address the impact of antibiotic treatment in sepsis outcome and discuss new insights for microbiota modulation." @default.
• W2472522918 created "2016-07-22" @default.
• W2472522918 creator A5015291257 @default.
• W2472522918 creator A5038683391 @default.
• W2472522918 creator A5053341542 @default.
• W2472522918 date "2016-07-01" @default.
• W2472522918 modified "2023-10-12" @default.
• W2472522918 title "The interplay between microbiota and inflammation: lessons from peritonitis and sepsis" @default.
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• W2472522918 doi "https://doi.org/10.1038/cti.2016.32" @default.
• W2472522918 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4973320" @default.
• W2472522918 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/27525063" @default.
• W2472522918 hasPublicationYear "2016" @default.
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