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- W2486337511 abstract "Since the carcinogenicity of BHA in rodent forestomach was first shown, several plant phenols have been demonstrated to exert carcinogenic potential in rats, e.g., caffeic acid (forestomach and kidney), sesamol (forestomach), catechol (glandular stomach) and hydroquinone (kidney). In the second stage of 2-stage carcinogenesis, these compounds demonstrate different modifying activities depending on the organ. For example, catechol enhances forestomach, glandular stomach, tongue and esophageal carcinogenesis while inhibiting colon, lung and hepato-carcinogenesis. Hydroquinone and caffeic acid enhance kidney and forestomach carcinogenesis, respectively. Several other phenolic antioxidants have been examined for their modifying effects using multi-organ carcinogenesis models. The results indicate that catechins clearly inhibit rat small intestinal carcinogenesis with the effects being most obvious when applied during the initiation stage. On the other hand, combined treatment with sodium nitrite and phenolic compounds such as catechol and 3-methoxycatechol strongly induces forestomach proliferative lesions which are not observed with individual treatments. The results suggest forestomach carcinogenicity of these compounds in the presence of sodium nitrite." @default.
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- W2486337511 date "1992-07-23" @default.
- W2486337511 modified "2023-10-14" @default.
- W2486337511 title "Carcinogenicity and Modification of Carcinogenic Response by Plant Phenols" @default.
- W2486337511 doi "https://doi.org/10.1021/bk-1992-0507.ch020" @default.
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