FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W2488048468> ?p ?o ?g. }

• W2488048468 endingPage "127" @default.
• W2488048468 startingPage "122" @default.
• W2488048468 abstract "Background and aims Airflow limitation, i.e. reduced forced expiratory volume in 1-s (FEV1), is associated with increased prevalence of atherosclerosis, however, causal mechanisms remain elusive. The objective of the study was to determine if the association between airflow obstruction and markers of atherosclerosis is mediated by systemic inflammation. Methods 1154 subjects from the longitudinal AGES Reykjavik study were included. Population characteristics, systemic inflammation markers from blood (white blood cell counts (WBC) and level of C-reactive protein (CRP)) were compared between patients with and without airflow limitation defined by reduced FEV1 on spirometry. Atherosclerosis burden was quantified by measurements of coronary artery calcium, aortic arch and distal aortic calcification in addition to carotid intimal media thickness (CIMT). Results Subjects were split into four groups according to smoking status and whether airflow limitation was present. There was a higher overall burden of atherosclerosis in ever-smokers compared to never-smokers, and in individuals with airflow obstruction compared to individuals without airflow obstruction. After adjusting for population characteristics, Framingham cardiovascular risk factors and markers of systemic inflammation (WBC and CRP), there was a significantly increased aortic arch and distal aorta calcification and higher CIMT measurement in individuals with airflow obstruction compared to individuals without airflow obstruction. After adjusting for population characteristics, Framingham cardiovascular risk factors and markers of systemic inflammation (WBC and CRP), there was a significantly increased aortic arch and distal aorta calcification and higher CIMT measurement in individuals with airflow obstruction compared to individuals without airflow obstruction. Conclusions Systemic inflammation (WBC and CRP) does not appear to mediate the association between airflow limitation and atherosclerosis. Only airflow limitation and not systemic inflammation (WBC and CRP) appears to be an independent predictor of atherosclerosis." @default.
• W2488048468 created "2016-08-23" @default.
• W2488048468 creator A5000325789 @default.
• W2488048468 creator A5002081827 @default.
• W2488048468 creator A5018378907 @default.
• W2488048468 creator A5018722034 @default.
• W2488048468 creator A5021935809 @default.
• W2488048468 creator A5022360631 @default.
• W2488048468 creator A5067651781 @default.
• W2488048468 creator A5072295805 @default.
• W2488048468 creator A5091376137 @default.
• W2488048468 date "2016-09-01" @default.
• W2488048468 modified "2023-10-18" @default.
• W2488048468 title "Airflow obstruction, atherosclerosis and cardiovascular risk factors in the AGES Reykjavik study" @default.
• W2488048468 cites W1480729244 @default.
• W2488048468 cites W1606597770 @default.
• W2488048468 cites W1841047255 @default.
• W2488048468 cites W1911878470 @default.
• W2488048468 cites W1964322185 @default.
• W2488048468 cites W1991265445 @default.
• W2488048468 cites W2010943681 @default.
• W2488048468 cites W2014443718 @default.
• W2488048468 cites W2042080482 @default.
• W2488048468 cites W2054931962 @default.
• W2488048468 cites W2063058274 @default.
• W2488048468 cites W2094941678 @default.
• W2488048468 cites W2097333427 @default.
• W2488048468 cites W2104168490 @default.
• W2488048468 cites W2125940552 @default.
• W2488048468 cites W2127951128 @default.
• W2488048468 cites W2132683688 @default.
• W2488048468 cites W2142560075 @default.
• W2488048468 cites W2153785016 @default.
• W2488048468 cites W2155175490 @default.
• W2488048468 cites W2155767904 @default.
• W2488048468 cites W2156281464 @default.
• W2488048468 cites W2167830562 @default.
• W2488048468 cites W2217208273 @default.
• W2488048468 cites W2231361272 @default.
• W2488048468 cites W2332933086 @default.
• W2488048468 cites W2614242365 @default.
• W2488048468 doi "https://doi.org/10.1016/j.atherosclerosis.2016.07.919" @default.
• W2488048468 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5963694" @default.
• W2488048468 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/27522264" @default.
• W2488048468 hasPublicationYear "2016" @default.
• W2488048468 type Work @default.
• W2488048468 sameAs 2488048468 @default.
• W2488048468 citedByCount "8" @default.
• W2488048468 countsByYear W24880484682018 @default.
• W2488048468 countsByYear W24880484682019 @default.
• W2488048468 countsByYear W24880484682021 @default.
• W2488048468 countsByYear W24880484682023 @default.
• W2488048468 crossrefType "journal-article" @default.
• W2488048468 hasAuthorship W2488048468A5000325789 @default.
• W2488048468 hasAuthorship W2488048468A5002081827 @default.
• W2488048468 hasAuthorship W2488048468A5018378907 @default.
• W2488048468 hasAuthorship W2488048468A5018722034 @default.
• W2488048468 hasAuthorship W2488048468A5021935809 @default.
• W2488048468 hasAuthorship W2488048468A5022360631 @default.
• W2488048468 hasAuthorship W2488048468A5067651781 @default.
• W2488048468 hasAuthorship W2488048468A5072295805 @default.
• W2488048468 hasAuthorship W2488048468A5091376137 @default.
• W2488048468 hasBestOaLocation W24880484682 @default.
• W2488048468 hasConcept C116067010 @default.
• W2488048468 hasConcept C126322002 @default.
• W2488048468 hasConcept C127413603 @default.
• W2488048468 hasConcept C164705383 @default.
• W2488048468 hasConcept C2776042228 @default.
• W2488048468 hasConcept C2776252253 @default.
• W2488048468 hasConcept C2776914184 @default.
• W2488048468 hasConcept C2779980429 @default.
• W2488048468 hasConcept C2780333948 @default.
• W2488048468 hasConcept C2781285907 @default.
• W2488048468 hasConcept C2908647359 @default.
• W2488048468 hasConcept C71924100 @default.
• W2488048468 hasConcept C78519656 @default.
• W2488048468 hasConcept C99454951 @default.
• W2488048468 hasConceptScore W2488048468C116067010 @default.
• W2488048468 hasConceptScore W2488048468C126322002 @default.
• W2488048468 hasConceptScore W2488048468C127413603 @default.
• W2488048468 hasConceptScore W2488048468C164705383 @default.
• W2488048468 hasConceptScore W2488048468C2776042228 @default.
• W2488048468 hasConceptScore W2488048468C2776252253 @default.
• W2488048468 hasConceptScore W2488048468C2776914184 @default.
• W2488048468 hasConceptScore W2488048468C2779980429 @default.
• W2488048468 hasConceptScore W2488048468C2780333948 @default.
• W2488048468 hasConceptScore W2488048468C2781285907 @default.
• W2488048468 hasConceptScore W2488048468C2908647359 @default.
• W2488048468 hasConceptScore W2488048468C71924100 @default.
• W2488048468 hasConceptScore W2488048468C78519656 @default.
• W2488048468 hasConceptScore W2488048468C99454951 @default.
• W2488048468 hasLocation W24880484681 @default.
• W2488048468 hasLocation W24880484682 @default.
• W2488048468 hasLocation W24880484683 @default.
• W2488048468 hasLocation W24880484684 @default.
• W2488048468 hasOpenAccess W2488048468 @default.
• W2488048468 hasPrimaryLocation W24880484681 @default.
• W2488048468 hasRelatedWork W1599314481 @default.

• next