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• W2489223214 startingPage "e0004912" @default.
• W2489223214 abstract "Malaria parasites dramatically alter the rheological properties of infected red blood cells. In the case of Plasmodium vivax, the parasite rapidly decreases the shear elastic modulus of the invaded RBC, enabling it to avoid splenic clearance. This study highlights correlation between rosette formation and altered membrane deformability of P. vivax-infected erythrocytes, where the rosette-forming infected erythrocytes are significantly more rigid than their non-rosetting counterparts. The adhesion of normocytes to the PvIRBC is strong (mean binding force of 440pN) resulting in stable rosette formation even under high physiological shear flow stress. Rosetting may contribute to the sequestration of PvIRBC schizonts in the host microvasculature or spleen." @default.
• W2489223214 created "2016-08-23" @default.
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• W2489223214 date "2016-08-10" @default.
• W2489223214 modified "2023-10-15" @default.
• W2489223214 title "Rheopathologic Consequence of Plasmodium vivax Rosette Formation" @default.
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• W2489223214 doi "https://doi.org/10.1371/journal.pntd.0004912" @default.
• W2489223214 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4980013" @default.
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