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W2493281570 endingPage "1351" @default.
W2493281570 startingPage "1339" @default.
W2493281570 abstract "Tardive dyskinesia (TD) is a serious and potentially irreversible side effect of long-term exposure to antipsychotic medication characterized by involuntary trunk, limb and orofacial muscle movements. Various mechanisms have been proposed for the etiopathophysiology of antipsychotic-induced TD in schizophrenia patients with genetic factors playing a prominent role. Earlier association studies have focused on polymorphisms in CYP2D6, dopamine-, serotonin-, GABA- and glutamate genes. This review highlights recent advances in the genetic investigation of TD. Recent promising findings were obtained with the HSPG2, DPP6, MTNR1A, SLC18A2, PIP5K2A and CNR1 genes. More research, including collection of well-characterized samples, enhancement of genome-wide strategies, gene-gene interaction and epigenetic analyses, is needed before genetic tests with clinical utility can be made available for TD." @default.
W2493281570 created "2016-08-23" @default.
W2493281570 creator A5022065571 @default.
W2493281570 creator A5039783734 @default.
W2493281570 creator A5055333011 @default.
W2493281570 date "2016-08-01" @default.
W2493281570 modified "2023-09-26" @default.
W2493281570 title "Pharmacogenetics of tardive dyskinesia: an updated review of the literature" @default.
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