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- W2495340561 abstract "This chapter emphasizes xenobiotic-induced structural and biochemical changes that occur in the kidney and lower urinary tract. Kidney is often exposed to a higher concentration of xenobiotics than other tissues. Since most xenobiotics or their metabolites are excreted in the urine, the urothelium, particularly that lining the urinary bladder, is frequently a target for toxicity. Any process that interferes with the structural integrity of the glomerulus can result in abnormal loss of protein in the urine, predominantly albumin. Glomerular injury may be non-inflammatory or inflammatory. Affected glomeruli may be observed grossly in some cases. Histologic changes are characterized by: hypercellularity due to cellular proliferation or leukocytic infiltration; basement membrane thickening; and hyalinization and sclerosis. Membranous nephropathy is thought to be caused by immune-complex deposition in the glomerular tuft, and is the most common cause of nephrotic syndrome in adults. Repeated (chronic) injection of soluble antigens produces this lesion, characterized by subepithelial immune-complex deposition, recognized light microscopically with silver stains as spikes of argyrophilic material. In routine histologic sections, the capillary wall of the affected glomerulus is diffusely slightly thickened." @default.
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- W2495340561 date "2010-01-01" @default.
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- W2495340561 title "Kidney and Lower Urinary Tract" @default.
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- W2495340561 doi "https://doi.org/10.1016/b978-0-12-370469-6.00011-8" @default.
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