FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W2495557463> ?p ?o ?g. }

• W2495557463 endingPage "25" @default.
• W2495557463 startingPage "25" @default.
• W2495557463 abstract "Human antioxidant protein 1 (Atox1) is a small cytosolic protein with an essential role in copper homeostasis. Atox1 functions as a copper carrier facilitating copper transfer to the secretory pathway. This process is required for activation of copper dependent enzymes involved in neurotransmitter biosynthesis, iron efflux, neovascularization, wound healing, and regulation of blood pressure. Recently, new cellular roles for Atox1 have emerged. Changing levels of Atox1 were shown to modulate response to cancer therapies, contribute to inflammatory response, and protect cells against various oxidative stresses. It has also become apparent that the activity of Atox1 is tightly linked to the cellular redox status. In this review, we summarize biochemical information related to a dual role of Atox1 as a copper chaperone and an antioxidant. We discuss how these two activities could be linked and contribute to establishing the intracellular copper balance and functional identity of cells during differentiation." @default.
• W2495557463 created "2016-08-23" @default.
• W2495557463 creator A5061858624 @default.
• W2495557463 creator A5079179187 @default.
• W2495557463 date "2016-07-27" @default.
• W2495557463 modified "2023-10-16" @default.
• W2495557463 title "The Role of Copper Chaperone Atox1 in Coupling Redox Homeostasis to Intracellular Copper Distribution" @default.
• W2495557463 cites W1565248232 @default.
• W2495557463 cites W1587735356 @default.
• W2495557463 cites W1906309090 @default.
• W2495557463 cites W1963874808 @default.
• W2495557463 cites W1964670708 @default.
• W2495557463 cites W1965043464 @default.
• W2495557463 cites W1965777297 @default.
• W2495557463 cites W1965891907 @default.
• W2495557463 cites W1967868572 @default.
• W2495557463 cites W1967942184 @default.
• W2495557463 cites W1968210490 @default.
• W2495557463 cites W1968740095 @default.
• W2495557463 cites W1969004465 @default.
• W2495557463 cites W1972240596 @default.
• W2495557463 cites W1978880986 @default.
• W2495557463 cites W1980099457 @default.
• W2495557463 cites W1988835673 @default.
• W2495557463 cites W1990181521 @default.
• W2495557463 cites W1995565650 @default.
• W2495557463 cites W1996024251 @default.
• W2495557463 cites W1999357946 @default.
• W2495557463 cites W2001499411 @default.
• W2495557463 cites W2003705772 @default.
• W2495557463 cites W2004471846 @default.
• W2495557463 cites W2011095090 @default.
• W2495557463 cites W2011444292 @default.
• W2495557463 cites W2012390982 @default.
• W2495557463 cites W2023694696 @default.
• W2495557463 cites W2024301095 @default.
• W2495557463 cites W2026433593 @default.
• W2495557463 cites W2028347889 @default.
• W2495557463 cites W2029729041 @default.
• W2495557463 cites W2031935607 @default.
• W2495557463 cites W2036363339 @default.
• W2495557463 cites W2037109359 @default.
• W2495557463 cites W2037119791 @default.
• W2495557463 cites W2043551256 @default.
• W2495557463 cites W2047523779 @default.
• W2495557463 cites W2049604444 @default.
• W2495557463 cites W2050610995 @default.
• W2495557463 cites W2051215316 @default.
• W2495557463 cites W2052078181 @default.
• W2495557463 cites W2052607444 @default.
• W2495557463 cites W2054006271 @default.
• W2495557463 cites W2054687549 @default.
• W2495557463 cites W2058861773 @default.
• W2495557463 cites W2066272870 @default.
• W2495557463 cites W2072383565 @default.
• W2495557463 cites W2076142778 @default.
• W2495557463 cites W2077116663 @default.
• W2495557463 cites W2078741049 @default.
• W2495557463 cites W2080026027 @default.
• W2495557463 cites W2083339401 @default.
• W2495557463 cites W2083615209 @default.
• W2495557463 cites W2085013311 @default.
• W2495557463 cites W2088537303 @default.
• W2495557463 cites W2091518477 @default.
• W2495557463 cites W2095320367 @default.
• W2495557463 cites W2104109524 @default.
• W2495557463 cites W2109512882 @default.
• W2495557463 cites W2114673327 @default.
• W2495557463 cites W2120194191 @default.
• W2495557463 cites W2134107421 @default.
• W2495557463 cites W2143131894 @default.
• W2495557463 cites W2143880121 @default.
• W2495557463 cites W2146421519 @default.
• W2495557463 cites W2146707901 @default.
• W2495557463 cites W2150850192 @default.
• W2495557463 cites W2156644611 @default.
• W2495557463 cites W2159517143 @default.
• W2495557463 cites W2165278512 @default.
• W2495557463 cites W2168911817 @default.
• W2495557463 cites W2193666963 @default.
• W2495557463 cites W2207695310 @default.
• W2495557463 cites W2228168283 @default.
• W2495557463 cites W2253955501 @default.
• W2495557463 cites W2256598572 @default.
• W2495557463 cites W2297411548 @default.
• W2495557463 cites W2321426138 @default.
• W2495557463 cites W4233005545 @default.
• W2495557463 cites W85469126 @default.
• W2495557463 cites W94808975 @default.
• W2495557463 doi "https://doi.org/10.3390/antiox5030025" @default.
• W2495557463 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5039574" @default.
• W2495557463 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/27472369" @default.
• W2495557463 hasPublicationYear "2016" @default.
• W2495557463 type Work @default.
• W2495557463 sameAs 2495557463 @default.
• W2495557463 citedByCount "83" @default.
• W2495557463 countsByYear W24955574632016 @default.
• W2495557463 countsByYear W24955574632017 @default.

• next