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- W2495998723 abstract "The classical picture of dopaminergic striatal synapses is simple and elegant. Nigro-striatal neurones release dopamine (DA) which activates inhibitory receptors on the striatal neurones. Parkinsonism would usually be the result of a deficit in striatal DA, and can be treated by replenishing the DA stores by the administration l-3,4-dihydroxyphenylalanine (l-DOPA). Here we present studies which suggest that 2-phenylethylamine and other endogenous compounds metabolically related to DA may modulate dopaminergic transmission at striatal synapses. The presence of endogenous phenylethylamine in brain was demonstrated by Sabelli, Giardina, Mosnaim and Sabelli (1973) and Inwang, Mosnaim and Sabelli (1973), and later confirmed by many others (see references in Sabelli, Mosnaim and Vazquez, 1974)." @default.
- W2495998723 created "2016-08-23" @default.
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- W2495998723 date "1976-01-01" @default.
- W2495998723 modified "2023-09-26" @default.
- W2495998723 title "Possible role of 2-phenylethylamine in the modulation of extrapyramidal function" @default.
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- W2495998723 doi "https://doi.org/10.1007/978-1-349-01247-3_15" @default.
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