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- W2497627842 endingPage "139" @default.
- W2497627842 startingPage "135" @default.
- W2497627842 abstract "This chapter focuses on the endothelin (ET) system, its components, and effects of preproet-1 or ET receptor gene deletion. ET production is modulated by inhibitors such as shear stress in blood vessels and nitric oxide (NO), and stimulators vasopressin, cytokines, insulin, and growth factors. ET receptors stimulate phospholipase C, inositol trisphosphate generation and calcium release, leading to calmodulin activation, diacylglycerol production and protein kinase C stimulation. In salt-dependent models of experimental hypertension such as DOCA-salt hypertension or Dahl salt-sensitive rats, and in severe hypertension such as stroke-prone spontaneously hypertensive rats (SHRsp), in particular when salt-loaded or treated with the nitric oxide synthase inhibitor L-NAME, enhanced production of ET-1. ETs are found in rostral portions of the brain, in the hypothalamus and the dorsal motor nucleus of the vagus and medulla oblongata as well as brainstem areas known to regulate cardiovascular function such as the area postrema, the ventrolateral medulla and the nucleus tractus solitarius." @default.
- W2497627842 created "2016-08-23" @default.
- W2497627842 creator A5046910928 @default.
- W2497627842 date "2012-01-01" @default.
- W2497627842 modified "2023-09-24" @default.
- W2497627842 title "The Endothelin System" @default.
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- W2497627842 doi "https://doi.org/10.1016/b978-0-12-386525-0.00028-7" @default.