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• W2497805638 abstract "Dysregulation of the stress response is implicated in many psychopathologies. Data gathered over the past two decades have proposed a rather dualistic view of the central corticotropin-releasing factor (CRF)-urocortin system. Originally, it was thought that CRF/CRF receptor type 1 (CRFR1) signaling mediated stress-initiated effects and increased anxiety-like behavior, whereas activation of urocortins/CRFR2 ensured adequate recovery from stress and restoration of homeostasis. However, this view was based on data gained from genetically modified mouse models and pharmacological approaches; now, with the emergence of new and more specific biological tools, it has become clear that this is an over-simplistic proposal. It is becoming apparent that the function of the CRF-urocortin system’s components relies profoundly on the spatial and temporal patterns of activity of the CRF family members. Here, we provide an overview of recent work that proposes a more dynamic, modulatory role for the CRF system’s central pathways in the modulation of stress-linked behaviors. Recent findings suggest that the CRF system’s actions are brain-region specific and dependent on the type of neuronal cell involved." @default.
• W2497805638 created "2016-08-23" @default.
• W2497805638 creator A5066695484 @default.
• W2497805638 date "2016-01-01" @default.
• W2497805638 modified "2023-10-18" @default.
• W2497805638 title "Genetic Dissection of the Neuroendocrine and Behavioral Responses to Stressful Challenges" @default.
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• W2497805638 doi "https://doi.org/10.1007/978-3-319-41603-8_6" @default.
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