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- W2499631282 abstract "Publisher Summary This chapter reviews the current clinical, histologic, and immunologic features of the most common forms of autoimmune bullous diseases. Autoimmune bullous diseases are rare disorders affecting skin and mucous membranes and are mediated by pathogenic autoantibodies against desmosomal or hemidesmosomal antigens of squamous epithelium. In the epidermis, neighboring keratinocytes adhere to each other through organelles known as desmosomes, whereas at the dermoepidermal junction hemidesmosomes anchor the epidermis to the dermis. The majority of these antigens recognized by these autoantibodies are desmosomal and hemidesmosomal transmembrane glycoproteins involved in epidermal cell–cell and epidermal–dermal adherence. The pemphigus syndromes include diseases that are characterized by loss of epidermal cell–cell cohesion and autoantibodies against desmosomal cadherins. This group comprises the two classical forms of pemphigus: pemphigus vulgaris (PV) and pemphigus foliaceus (PF). PV is characterized by suprabasilar acantholysis and anti-Dsg3 IgG autoantibodies, whereas PF is characterized by subcorneal acantholysis and anti-Dsg1 IgG autoantibodies. Other infrequent forms of pemphigus include paraneoplastic pemphigus (PNP), drug-induced pemphigus, and IgA pemphigus are also discussed." @default.
- W2499631282 created "2016-08-23" @default.
- W2499631282 creator A5006935079 @default.
- W2499631282 creator A5050789569 @default.
- W2499631282 creator A5056400278 @default.
- W2499631282 creator A5075875145 @default.
- W2499631282 date "2006-01-01" @default.
- W2499631282 modified "2023-09-26" @default.
- W2499631282 title "Bullous Skin Diseases: Pemphigus Pemphigoid" @default.
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- W2499631282 doi "https://doi.org/10.1016/b978-012595961-2/50060-3" @default.
- W2499631282 hasPublicationYear "2006" @default.