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- W25012202 abstract "New genetic fate-mapping technology in mice, neuroimaging and identification of several genes involved in human congenital cerebellar diseases have greatly improved our understanding of cerebellar development and pathogenesis. The pattern of expression of connexins in the cerebellum overlaps with the set of connexins expressed in the entire central nervous system (CNS). Because the cerebellum is not the primary brain structure to be scrutinized in the absence of obvious signs of cerebellar impairment in engineered connexin-gene mice, the role of connexins in the developing and adult cerebellum is probably underestimated. Some connexin-related pathology of the cerebellum has been identified in syndromes that are associated with dysfunctions of other brain and organ structures. Connexins expressed by glial cells, astrocytes and oligodendrocytes during development have been identified as important mediators of cerebellar impairment. Studies on the expression of glial connexins, especially astrocytic connexins, may broaden our understanding of the functional and pathological roles of gap junctions in the CNS and cerebellum." @default.
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- W25012202 date "2013-01-01" @default.
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- W25012202 title "Gap Junctions in Cerebellar Development and Pathology" @default.
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