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- W2505385575 abstract "isothiourea (SMT) or N G -nitro-L-arginine methyl ester chain enzymes and gluconeogenesis. 5 In cultured hepato- (L-NAME). iNOS induction was assessed by Western cytes, NO inhibits total protein synthesis 6 and bile cana- blot, immunohistochemistry, and measurement of NO licular contraction. 7 NO has been implicated in several metabolites in plasma and bile. Liver damage was de- pathophysiological processes such as endotoxemia (sep- termined by aspartate aminotransferase and alanine sis), hepatic ischemia/reperfusion injury, and hepatic allo- aminotransferase and by histology. The effects of both graft rejection. 8 However, the exact function of iNOS inhibitors on systemic and portal pressure were mea- and enhanced NO production under these conditions has sured in normal and LPS-treated rats. Results: LPS not been elucidated yet. Most studies postulate a hepato- treatment strongly induced iNOS in inflammatory cells," @default.
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- W2505385575 date "1997-10-01" @default.
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- W2505385575 title "Differential Effects of Nitric Oxide Synthase Inhibitors on Endotoxin-Induced Liver Damage in Rats" @default.
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