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• W2507772654 abstract "Innate immunity plays a crucial role in the pathogenesis of type 2 diabetes and related complications. Since the toll-like receptors (TLRs) are central to innate immunity, it appears that they are important participants in the development and pathogenesis of the disease. Previous investigations demonstrated that TLR2 homodimers and TLR2 heterodimers with TLR1 or TLR6 activate innate immunity upon recognition of damage-associated molecular patterns (DAMPs). Several DAMPs are released during type 2 diabetes, so it may be hypothesized that TLR2 is significantly involved in its progression. Here, we review recent data on the important roles and status of TLR2 in type 2 diabetes and related complications." @default.
• W2507772654 created "2016-09-16" @default.
• W2507772654 creator A5010605049 @default.
• W2507772654 creator A5015337593 @default.
• W2507772654 creator A5026402543 @default.
• W2507772654 creator A5064065937 @default.
• W2507772654 date "2016-07-28" @default.
• W2507772654 modified "2023-10-13" @default.
• W2507772654 title "Toll-like receptor 2 and type 2 diabetes" @default.
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• W2507772654 doi "https://doi.org/10.1186/s11658-016-0002-4" @default.
• W2507772654 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5415836" @default.
• W2507772654 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/28536605" @default.
• W2507772654 hasPublicationYear "2016" @default.
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