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- W2511769965 endingPage "120" @default.
- W2511769965 startingPage "112" @default.
- W2511769965 abstract "The interaction of neutrophils (PMNs) and epithelial cells are requisite lines of communication during mucosal inflammatory responses. Consequences of such interactions often determine endpoint organ function, and for this reason, much interest has developed around defining the constituents of the tissue microenvironment of inflammatory lesions. Physiologic in vitro and in vivo models have aided in the discovery of components that define the basic inflammatory machinery that mold the inflammatory tissue microenvironment. Here, we will review the recent literature related to the contribution of PMNs to molding of the tissue microenvironment, with an emphasis on the gastrointestinal (GI) tract. We focus on endogenous pathways for promoting tissue homeostasis and the molecular determinants of neutrophil-epithelial cell interactions during ongoing inflammation. These recent studies highlight the dynamic nature of these pathways and lend insight into the complexity of treating mucosal inflammation." @default.
- W2511769965 created "2016-09-16" @default.
- W2511769965 creator A5037858531 @default.
- W2511769965 creator A5049650132 @default.
- W2511769965 creator A5065279844 @default.
- W2511769965 date "2016-08-25" @default.
- W2511769965 modified "2023-10-18" @default.
- W2511769965 title "Neutrophils and the inflammatory tissue microenvironment in the mucosa" @default.
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