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- W2512551368 abstract "Spinal cord injury (SCI) causes rapid, severe osteoporosis with increased fracture risk. The pathogenesis of osteoporosis after SCI is a complex process and is usually attributed to “disuse” or “immobilization.” However, the exact pathophysiology of osteoporosis after SCI is still not clear. In SCI, bone remodeling becomes uncoupled with an initial decrease in bone formation and steadily increasing bone resorption. Osteoporosis after SCI can be evaluated by measuring BMD using DEXA, pQCT, and MRI; and estimating biochemical markers of bone turnover. Bone cell activity can be evaluated indirectly with techniques, such as specific serum and urine biochemical markers of bone turnover. An improved understanding of the natural history and risk factors for chronic bone loss following SCI is essential to designing therapies to reduce the rate of bone loss, define fracture risk, and ultimately prevent osteoporotic fractures and their associated morbidity. In conclusion, we are of the opinion that prospective randomized controlled trials should be conducted to evaluate, standardize, and find bone-specific biochemical marker of bone turnover, for the better understanding of the pathophysiology of osteoporosis in SCI." @default.
- W2512551368 created "2016-09-16" @default.
- W2512551368 creator A5088787798 @default.
- W2512551368 date "2017-01-01" @default.
- W2512551368 modified "2023-09-27" @default.
- W2512551368 title "Bone Turnover and Spinal Cord Injury" @default.
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