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• W2512796610 abstract "Failure of gut homeostasis is an important factor in the pathogenesis and progression of systemic inflammation, which can culminate in multiple organ failure and fatality. Pathogenic events in critically ill patients include mesenteric hypoperfusion, dysregulation of gut motility, and failure of the gut barrier with resultant translocation of luminal substrates. This is followed by the exacerbation of local and systemic immune responses. All these events can contribute to pathogenic crosstalk between the gut, circulating cells, and other organs like the liver, pancreas, and lungs. Here we review recent insights into the identity of the cellular and biochemical players from the gut that have key roles in the pathogenic turn of events in these organ systems that derange the systemic inflammatory homeostasis. In particular, we discuss the dangers from within the gastrointestinal tract, including metabolic products from the liver (bile acids), digestive enzymes produced by the pancreas, and inflammatory components of the mesenteric lymph." @default.
• W2512796610 created "2016-09-16" @default.
• W2512796610 creator A5000603064 @default.
• W2512796610 creator A5010721047 @default.
• W2512796610 creator A5067048349 @default.
• W2512796610 date "2016-10-18" @default.
• W2512796610 modified "2023-09-30" @default.
• W2512796610 title "The digestive tract as the origin of systemic inflammation" @default.
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