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- W2514467810 abstract "Contexte les CE/PN s’accompagnent frequemment d’un hypercortisolisme severe et une hypokaliemie. Objectif Preciser les mecanismes de l’hypokaliemie et l’implication du SRA et autres M. Patients A partir de 162 dossiers de Cushing ACTH dependants, classes CE/PN (periode 2008–2016), ont ete inclus 89 avec des donnees suffisantes (F : 47 ; H : 42) dont CE/PN certain ( n = 64) (=anapath TNE/immunohisto ACTH + , ou KT-SPI pas de gradient + IRM hypophyse nle) ou probables ( n = 25) (tests dynamiques neg + IRM-h nle + TNE visible ou non). Methodes Furent extraits : traitements, PA, kaliemie (K + )/kaliurese ; CLU (RIAs ou LCMS), ACTH (≠sandwichs) ; desoxycortecosterone (DOC), dosages : extraction/RIAs, extraction/chromato/RIAs ou LCMS), aldosterone (RIAs/LCMS), Renine (ECLIAs, RIAs), THF + 5αTHF/THE (urines, GCMS). Resultats (Mediane-range) : HTA : n = 85 ; avant tout traitement anticortisolique ou perturbant SRA : kaliemie/mmol/L, n = 69 : 2,8 (1,3–3,9) et kaliurese/mmol/24 h, n = 14 : 41,5 (24–73) ; CLU/μg/24 h, n = 89 : 1614 (294–20 220) ; ACTH (pg/ml) ( n = 89 : 143 (24–922) ; Etaient en dessous limite inferieure de la normale, pour DOC : 16/27 ; Aldosterone : 28/38 ; Renine 29/37 et chez aucun patient Aldosterone ou DOC elevee ; ratio THF + 5αTHF/THE ( n = 8) eleves : 4,2 (1,3–6,8). Correlation significative entre K + et CLU (r = -0,5859. p n = 51). Conclusions L’hypokaliemie des CE/PN, est due a une fuite urinaire de potassium causee par l’augmentation majeure du cortisol, insuffisamment inactive en cortisone et agissant comme principal M, d’ou l’effet de la spironolactone. La DOC ou le SRA ne sont pas impliques." @default.
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- W2514467810 date "2016-09-01" @default.
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- W2514467810 title "Cushing ectopique/paranéoplasique (CE/PN) : métabolisme du potassium, minéralocorticoïdes (M) et système rénine-aldostérone (SRA). Étude multicentrique française (EctoCush-France)" @default.
- W2514467810 doi "https://doi.org/10.1016/j.ando.2016.07.070" @default.
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