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- W2519093800 abstract "Diabetic Nephropathy (DN) is considered the main cause of end stage kidney disease around the world. However, its pathogenesis is not completely established. More than just a direct consequence of chronic glycemic changes, recent studies had suggested Diabetic Nephropathy could be considered an inflammatory disease. It has been shown that concentrations of pro-inflammatory cytokines, as IL-1, IL-6, IL-18, IL-33, IFN-γ and TNF-α actively participate in development and progression of DN, and thus, are involved in pathogenesis. Besides, changes in acquired immune response, especially the presence of cellular immune response profiles of pro-inflammatory and effector nature, mainly Th1 and Th17, as the imbalance between interaction of cytokines and T regulatory cells, foment the onset and progression of DN. Here we summarize the main evidences that support the critical role of the immune system in this condition. These new conceptual advances in DN understanding are essential for development of new the rapeutical strategies and prognostic factors, which could be protagonists or adjuvants to the current ones, leading ultimately to a better clinical management of DN patients." @default.
- W2519093800 created "2016-09-23" @default.
- W2519093800 creator A5005852691 @default.
- W2519093800 creator A5008230762 @default.
- W2519093800 creator A5012944084 @default.
- W2519093800 creator A5018936499 @default.
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- W2519093800 creator A5053242133 @default.
- W2519093800 creator A5079228065 @default.
- W2519093800 creator A5081853518 @default.
- W2519093800 date "2016-01-01" @default.
- W2519093800 modified "2023-10-12" @default.
- W2519093800 title "Cytokines and T Helper Cells in Diabetic Nephropathy Pathogenesis" @default.
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