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- W2526163621 abstract "β-endorphin (β-end), methionin-enkephalin (Met-enk) and noradrenaline (Norad) in the plasma and spinal fluid of patients with cerebral vascular disease (CVD) were measured at intervals from the onset of disease, and opioid peptide during CVD was studied. During CVD, β-end level in the spinal fluid was elevated significantly, but, this elevation was not detected in the plasma, suggesting that it was due to advance in production or secretion of the substance by CNS cells.In addition, it was suspected that the tissue Na ion level was elevated due to ischemic cerebral edema as a consequence of CVD, probably causing the changes in the structure of opioid receptor by allosteric effect resulting in the inhibition of binding/breakdown of β-end and its eventual release into the spinal fluid.Plasma Met-enk, which is present in the same granule as Norad within the adrenal cells, also increased at the time of onset of CVD and again increased during the acute phase of the disease, showing a significant negative correlation with plasma Norad during and after the subacute phase of cerebral hemorrhage, suggesting that opioid peptide functions as a feedback mechanism in the control of the autonomic nervous system during CVD." @default.
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- W2526163621 date "1988-01-01" @default.
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- W2526163621 title "Studies of .BETA.-endorphin and methionin-enkephalin in cerebral vascular disease. Its evaluation and clinical significance of periodic changes." @default.
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- W2526163621 doi "https://doi.org/10.1272/jnms1923.55.380" @default.
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