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- W252645398 abstract "To explore the mechanism of swine edema,the cultured rat intestinal microvascular endothelial cells(RIMECs) were divided into control group and different concentration of SLT-Ⅱe groups.Secretions of NO,ET-1,P-selectin,sICAM-1 and TNF-α by RIMECs were determined by ELISA at 3,6,9,and 12 h post-induction.The results showed that 1 and 10 μg/mL SLT-Ⅱe increased the NO production in RIMECs by 3 times for 12 h incubation.1 μg/mL SLT-Ⅱe could increase the secretion of NO in RIMECs by 3 times,ET-1,P-selectin,sICAM-1and TNF-α by 4.6,2.8,2.8 amd 1.8 times for 12 h,compared with control group,respectively.The results revealed that SLT-Ⅱe caused swine edema by over inducing the secretion of NO,ET-1,P-selectin,sICAM-1and TNF-α in RIMECs,and dramatically decreasing the NO/ET-1 ratio,which might result in intestinal microcirculation obstruction.The best model concentration of SLT-Ⅱe to damage the RIMECs was found to be 1 μg/mL." @default.
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- W252645398 date "2009-01-01" @default.
- W252645398 modified "2023-09-26" @default.
- W252645398 title "Effects of SLT-II e on the secretion of NO, ET-1, P-selectin, sICAM-1 and TNF-α in rat intestinal microvascular endothelial cells." @default.
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