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• W2527338241 abstract "Diabetic patients have increased fracture risk. The pathogenesis underlying the status of bone alterations in diabetes mellitus is not completely understood but is multifactorial. The major deficits appear to be related to a deficit in mineralized surface area, a decrement in the rate of mineral apposition, deceased osteoid surface, depressed osteoblast activity, and decreased numbers of osteoclasts due to abnormal insulin signaling pathway. Other prominent features of diabetes mellitus are an increased urinary excretion of calcium and magnesium, accumulation of advanced glycation end products, and oxidative stress leading to sweet bones (altered bone’s strength, metabolism, and structure). Every diabetic patient should be assessed for risk factors for fractures and osteoporosis. The pathogenesis of the bone alterations in diabetes mellitus as well as their molecular mechanisms needs further study." @default.
• W2527338241 created "2016-10-07" @default.
• W2527338241 creator A5074793027 @default.
• W2527338241 date "2016-01-01" @default.
• W2527338241 modified "2023-09-25" @default.
• W2527338241 title "Sweet Bones: The Pathogenesis of Bone Alteration in Diabetes" @default.
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• W2527338241 doi "https://doi.org/10.1155/2016/6969040" @default.
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