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- W252990054 abstract "In response to brain ischemia a cascade of signals leads to the activation of the brain innate immune system and to the recruitment of blood borne derived cells to the ischemic tissue. These processes have been increasingly shown to play a role on stroke pathogenesis. Here, we discuss the key features of resident microglia and different leukocyte subsets implicated in cerebral ischemia with special emphasis of neutrophils, monocytes and microglia. We focus on how leukocytes are recruited to injured brain through a complex interplay between endothelial cells, platelets and leukocytes and describe different strategies used to inhibit their recruitment. Finally, we discuss the possible existence of different leukocyte subsets in the ischemic tissue and the repercussion of different myeloid phenotypes on stroke outcome. The knowledge of the nature of these heterogeneous cell-cell interactions may open new lines of investigation on new therapies to promote protective immune responses and tissue repair after cerebral ischemia or to block harmful responses. This article is part of a Special Issue entitled SI: Cell Interactions In Stroke." @default.
- W252990054 created "2016-06-24" @default.
- W252990054 creator A5016967825 @default.
- W252990054 creator A5020184761 @default.
- W252990054 creator A5055174431 @default.
- W252990054 creator A5091527761 @default.
- W252990054 date "2015-10-01" @default.
- W252990054 modified "2023-10-18" @default.
- W252990054 title "Complexity of the cell–cell interactions in the innate immune response after cerebral ischemia" @default.
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