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• W2530913444 startingPage "263" @default.
• W2530913444 abstract "RAS mutations are among the most common genetic alterations found in cancerous tumors but rational criteria or strategies for targeting RAS-dependent tumors are only recently emerging. Clinical and laboratory data suggest that patient selection based on specific RAS mutations will be an essential component of these strategies. A thorough understanding of the biochemical and structural properties of mutant RAS proteins form the theoretical basis for these approaches. Direct inhibition of KRAS G12C by covalent inhibitors is a notable recent example of the RAS mutation-tailored approach that establishes a paradigm for other RAS mutation-centered strategies." @default.
• W2530913444 created "2016-10-21" @default.
• W2530913444 creator A5034326513 @default.
• W2530913444 creator A5037600867 @default.
• W2530913444 creator A5064122269 @default.
• W2530913444 date "2017-02-01" @default.
• W2530913444 modified "2023-10-14" @default.
• W2530913444 title "Rationale for <i>RAS</i> mutation-tailored therapies" @default.
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• W2530913444 doi "https://doi.org/10.2217/fon-2016-0363" @default.
• W2530913444 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/27728979" @default.
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