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- W2531894945 abstract "Background/Aim: Approximately 20 – 25% of ovarian cancers are attributable to germline or somatic BRCA1/2 mutations, resulting in defects in the homologous recombination (HR) pathway. Inactivation of these genes can also be mediated by epigenetic changes e.g. hypermethylation of CpG islands in the promoter regions. Aim of this analysis was to evaluate the stability of BRCA1 promoter hypermethylation in recurrent disease after platinum based chemotherapy." @default.
- W2531894945 created "2016-10-21" @default.
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- W2531894945 date "2016-10-13" @default.
- W2531894945 modified "2023-09-27" @default.
- W2531894945 title "Loss of BRCA1 promotor hypermethylation in recurrent high grade ovarian cancer" @default.
- W2531894945 doi "https://doi.org/10.1055/s-0036-1592709" @default.
- W2531894945 hasPublicationYear "2016" @default.
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