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- W2533148927 abstract "Although amnestic mild cognitive impairment (aMCI) with high cerebral deposition of beta-amyloid protein (Aβ) could be classified as in prodromal state of Alzheimer’s disease (AD) dementia, aMCI with the lack of or very little cerebral Aβ deposition is likely related to other pathophysiological processes. We aimed to investigate the differential patterns of regional cerebral glucose metabolism (rCGM) according to the level of brain Aβ burden in aMCI. Twenty-five aMCI and 33 cognitive normal (CN) elderly underwent comprehensive clinical examination and multi-modal brain imaging including 11C-labelled Pittsburgh Compound B (PiB) positron emission tomography (PET), 18F-deoxyglucose PET and magnetic resonance imaging. Based on PiB retention, the aMCI subjects were divided into low Aβ (aMCI-, n=10) and high Aβ (aMCI+, n=15) subgroups. Differences in rCGM among CN and aMCI subgroups were estimated on a voxel by voxel basis. When compared to CN, aMCI- showed lower rCGM only in the bilateral hippocampi, while aMCI+ had decreased rCGM in the bilateral hippocampi and right precuneus as expected. Additionally, aMCI+ individuals showed lower rCGM in the right precuneus than aMCI-." @default.
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- W2533148927 date "2016-07-01" @default.
- W2533148927 modified "2023-10-14" @default.
- W2533148927 title "P2-256: Differential Regional Cerebral Hypometabolism Patterns According to the Level of Cerebral Amyloid Deposition in Mild Cognitive Impairment" @default.
- W2533148927 doi "https://doi.org/10.1016/j.jalz.2016.06.1425" @default.
- W2533148927 hasPublicationYear "2016" @default.
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