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- W2549171330 abstract "<b>Background and Objective:</b> Biomass causes lung inflammation and parenchymal collagen tissue destruction like emphysema. Serum Prolidase enzyme activity is correlated with increased rates of collagen turnover. We firstly assessed the relation between prolidase activity and and its association with MPO as a marker of neutrophilic inflammation in healthy biomass exposed women. <b>Methods:</b> A total of 105 women were enrolled in this study. We measured serum prolidase enzyme activity (SPEA) and Myeloperoxidase (MPO). We also investigated oxidative–antioxidative status in these groups. <b>Results:</b> Significantly higher SPEA and MPO were found in the HBE group compared with the COPD and control groups (p < 0.05, p = 0. 01). In the biomass-exposed groups, the SPEA was positively correlated with the MPO level (r = 0.237, p = 0.04). <b>Conclusions:</b> elevated SPEA may be related to neutrophilic inflammation. SPEA may also reflect elevated collagen tissue metabolism and turnover in the lung. It may therefore serve as a marker of the progression of lung disease in these cases. However, this is the first study on this association. Further studies should be undertaken to confirm our findings." @default.
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- W2549171330 date "2016-09-01" @default.
- W2549171330 modified "2023-10-04" @default.
- W2549171330 title "Does prolidase activity an early marker for lung emphysema in biomass smoke exposured women" @default.
- W2549171330 doi "https://doi.org/10.1183/13993003.congress-2016.pa4295" @default.
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