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- W2552573409 endingPage "13" @default.
- W2552573409 startingPage "2" @default.
- W2552573409 abstract "Obesity is a major burden on healthcare systems worldwide due to the association with numerous complications, arguably the most important of which are the development of type 2 diabetes and cardiovascular disease. Both are thought to develop from similar origins and occur at variable rates in obese individuals, including those with similar body mass indices. This phenomenon is likely a result of an increased susceptibility for the storage of excess fat in the wrong place, namely, ectopic fat surrounding the liver, pancreas and muscles. This triggers a concatenation of events leading to insulin resistance and inflammation which culminate in an increased atherothrombotic potential due to the dysfunction of vascular endothelial cells causing accelerated atherosclerotic plaque formation and a pro-thrombotic phenotype. The degree of weight loss following different interventions is well documented but it is less widely known what effect weight loss by various means has on the deleterious process mentioned above, in particular their effects on cardiovascular events. This review summarises the processes leading to increased vascular risk in obesity and examines the effects of currently available weight loss strategies on reversing these processes and how this translates to cardiovascular disease." @default.
- W2552573409 created "2016-11-30" @default.
- W2552573409 creator A5007419681 @default.
- W2552573409 creator A5086223086 @default.
- W2552573409 date "2016-11-14" @default.
- W2552573409 modified "2023-10-17" @default.
- W2552573409 title "Vascular risk in obesity: Facts, misconceptions and the unknown" @default.
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