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- W2553621285 abstract "D-Asp is highly concentrated in the prenatal brain; after birth, its levels sharply decrease due to the catabolising activity of the enzyme D-aspartate oxidase. D-Asp is able to stimulate NMDAR-dependent neurotransmission through direct action at the glutamate-binding site of NMDARs, thus functioning as an endogenous agonist for this subclass of glutamate receptors. The agonistic activity exerted by D-Asp on NMDARs and its neurodevelopmental occurrence make this D-amino acid a potential mediator for some of the NMDAR-related alterations observed in schizophrenia. Therefore, aim of the research is to investigate about the potential role of D-Asp in schizophrenia-relevant circuits and behaviors." @default.
- W2553621285 created "2016-11-30" @default.
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- W2553621285 date "2016-03-29" @default.
- W2553621285 modified "2023-09-23" @default.
- W2553621285 title "Biological function of D-aspartate in the mammalian brain" @default.
- W2553621285 hasPublicationYear "2016" @default.
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