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- W2554219892 abstract "Autophagy is known as an evolutionarily conserved cellular degradation pathway for the recycling of cytoplasmic organelles and proteins. The autophagic pathway is also stimulated by various stresses, such as starvation, oxidative stress and pathogen infection. Autophagy serves to a cellular defense mechanism against pathogens. Epstein-Barr Virus (EBV) is an oncogenic virus, associated with various malignancies of epithelial and lymphoid origin, in which the majority of cells harbor a latent infection in vivo. Increasing evidence has shown that the host cells try to control EBV infection by activating the autophagic machinery. EBV has evolved novel ways of modulating autophagy during latent and lytic phases of the virus life cycle. In this review, we present an overview of recent advances in our understanding of the complex interplay between autophagy and EBV. The comprehensive understanding of the interactions between autophagy and EBV may lead to novel therapeutic strategies against EBV-related malignancies." @default.
- W2554219892 created "2016-11-30" @default.
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- W2554219892 date "2015-01-01" @default.
- W2554219892 modified "2023-10-12" @default.
- W2554219892 title "Interaction between Autophagy and EBV: Impacts on the Autophagic Pathway and EBV Infection" @default.
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