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- W2555694732 endingPage "17" @default.
- W2555694732 startingPage "11" @default.
- W2555694732 abstract "Cardiotoxicity induced by chemotherapeutic agents and radiotherapy is a growing problem. In recent years, an increasing number of new drugs with targeted action have been designed. These molecules, such as monoclonal antibodies and tyrosine kinase inhibitors, can cause different type of toxicities compared to traditional chemotherapy. However, they can also cause cardiac complications such as heart failure, arterial hypertension, QT interval prolongation and arrhythmias. Currently, a field of intense research is the vascular toxicity induced by new biologic drugs, particularly those which inhibit vascular endothelial growth factor (VEGF) and its receptor (VEGF-R) and other tyrosine kinases. In this review, we aim at focusing on the problem of vascular toxicity induced by new targeted therapies, chemotherapy and radiotherapy, and describe the main mechanisms and emphasizing the importance of early diagnosis of vascular damage, in order to prevent clinical complications." @default.
- W2555694732 created "2016-11-30" @default.
- W2555694732 creator A5013180336 @default.
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- W2555694732 creator A5053611466 @default.
- W2555694732 creator A5056329331 @default.
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- W2555694732 creator A5059792968 @default.
- W2555694732 creator A5061316252 @default.
- W2555694732 creator A5076173791 @default.
- W2555694732 date "2017-01-01" @default.
- W2555694732 modified "2023-09-29" @default.
- W2555694732 title "Anticancer therapy-induced vascular toxicity: VEGF inhibition and beyond" @default.
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