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- W2555750048 abstract "Introduction: Obstructive sleep apnea syndrome is characterized by recurrent upper airway obstruction and CIH during sleep, but it is still unknown that what effect on upperairway could be caused by CIH and how this effect works. Objective: Recent study showed CIH could induce the increase of a1-adrenoceptor immunoactivity in the hypoglossal nucleus of the rats. This study aimed to explore whether norepinephrine is involved in the central regulation of genioglossus during CIH. Methods: 24 male Wistar rats were exposed to CIH and divided into CIH, CIH with drug (the a1-adrenoceptor antagonist prazosin) and CIH with NaCl. The CIH exposure was applied by hypoxic compartment and lasted 21days according to previous study. At the same time, 22 rats were exposed to the alternating cycles of air under identical experimental conditions as CIH in parallel. These rats are similarly divided into air, air with drug and air with NaCl. The drug and NaCl were microinjected to the hypoglossal nucleus located by the rat brain stereotaxic apparatus. Transcranial magnetic stimulation (TMS) was applied for every rat to record genioglossus corticomotor responses on the 1st, 7th, 14 th and 21st day of the experiment. Results: Compared to the air group, CIH group showed shortened TMS latencies (P Conclusion: Norepinephrine participated in the central regulation of genioglossus during CIH in rats." @default.
- W2555750048 created "2016-11-30" @default.
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- W2555750048 date "2016-09-01" @default.
- W2555750048 modified "2023-09-27" @default.
- W2555750048 title "Dose norephinephrine participate in the central regulation of genioglossus in chronic-intermittent-hypoxia rat?" @default.
- W2555750048 doi "https://doi.org/10.1183/13993003.congress-2016.oa4560" @default.
- W2555750048 hasPublicationYear "2016" @default.
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