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- W2556559214 abstract "Copyright: © 2014 Scicchitano P, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Cardiovascular diseases are the leading cause of death in the Western countries despite the improvement in the diagnosis and treatment of affected patients [1]. Atherosclerotic process and its complications are the main determinants of cardiovascular diseases’ development. In particular, the development of an inflammatory, systemic condition of the vessels is able to increase the generation of the atherosclerotic plaques and makes them to become unstable and more prone to rupture [1,2]. Although histology and cytology has already explained the inner determinants of the atherosclerotic plaque, the cells implicated in the negative and positive remodeling of the vascular walls and the inflammatory conditions within the plaques, some point still remain obscure [2-4]. In particular, the receptor activator of nuclear factor(NF)κB ligand (RANKL)/receptor activator of nuclear factor (NF) κB (RANK)/osteoprotegerin (OPG) system has emerged as playing a key role in the genesis of atherosclerotic plaque composition and intima arterial calcification [2,3,5]." @default.
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- W2556559214 date "2014-01-01" @default.
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- W2556559214 title "The Role of Intimal Arterial Calcification in the Context of Atherosclerotic Plaque Stability Pietro Scicchitano1*, Andrea Marzullo2" @default.
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- W2556559214 doi "https://doi.org/10.4172/2157-7099.1000e111" @default.
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