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• W2556728638 abstract "<b><i>Background:</i></b> Post-stroke fatigue (PSF) is a common sequela of stroke. Despite reports of serotonergic involvement in the etiology of PSF, the potential contribution of serotonergic genes in the development of PSF needs to be investigated. <b><i>Methods:</i></b> A total of 373 patients, who experienced ischemic stroke for PSF, were evaluated 3 months after the stroke. PSF was assessed using the Fatigue Severity Scale. The genomic DNA collected and stored in a -70°C freezer was genotyped for 6 polymorphisms in genes associated with serotonin synthesis (tryptophan hydroxylase 1 (TPH1) A218C, TPH2 rs10879355, and TPH2 rs4641528), transport (the promoter region of the serotonin transporter protein), and catabolism (the 30-bp functional variable number tandem repeat) polymorphism in the promoter region of <i>monoamine oxidase A (MAO-A)</i>. <b><i>Results:</i></b> Among the 373 patients, 164 (44%) had PSF. All patients were ethnic Koreans. Of the 6 polymorphisms examined, only one marker, that is, low-activity <i>MAO-A</i> was associated with PSF (p < 0.05) in female patients. Multiple logistic regression analyses showed that post-stroke depression (PSD; 95% CI 1.561-14.323, p = 0.006) and low <i>MAO-A</i> activity (95% CI 0.166-0.722, p = 0.005) were factors associated with PSF in female patients, whereas only PSD (95% CI 5.511-65.269, p = 0.000) was associated with PSF in male patients. <b><i>Conclusions:</i></b> Our findings suggest that PSF may be associated with a genetic polymorphism involving <i>MAO-A</i>, at least in female stroke patients." @default.
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• W2556728638 date "2016-11-19" @default.
• W2556728638 modified "2023-10-14" @default.
• W2556728638 title "Post-Stroke Fatigue May Be Associated with the Promoter Region of a <b><i>Monoamine Oxidase A </i></b>Gene Polymorphism" @default.
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• W2556728638 doi "https://doi.org/10.1159/000450894" @default.
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